Tonsil Cancer
Q. What are the tonsils?
Ans. The pharynx is the continuation of the nose and mouth. It is a muscular tube that continues downwards through the neck and is responsible for the passage of both air (to the larynx, trachea and lungs) and food (to the oesophagus and then stomach). The pathways for food and air cross over in the pharynx. In addition, the auditory canal opens onto the upper part of the pharynx.
The walls of the pharynx are composed of fascia and muscle layers all lined by a mucous membrane. The pharynx is divided into three different areas based on anatomical location: the nasopharynx (behind the nose); oropharynx (behind the mouth); and the laryngopharynx (behind the larynx).
The tonsils are a ring of lymphoid tissue around the upper part of the pharynx. They consist of the lingual tonsil in the posterior part of the tongue, the palatine tonsils and the pharyngeal tonsils. Lymphoid tissue acts as a barrier against infection.
Q. How is Tonsil Cancer treated?
Ans. Cancer-Healer is found to be effective in tonsillar cancers. where it brings marked improvement.
It brings prompt succour and relief in all patients and can also be effectively been cured if diagnosed early and prompt treatment.
Saturday, March 14, 2009
GALL BLADDER CANCER
GALL BLADDER CANCER
Q. What are the types of gallbladder cancer?
A. The majority of these cancers are "adenocarcinomas", with subtypes such as papillary, nodular, and tubular, depending on the appearance of the tumor cells under the microscope. Less common subtypes include: squamous cell, signet ring cell, and adenosquamous (adenoacanthoma).
Q. What causes gallbladder cancer? What are the risk factors?
A. The cause of gallbladder cancer is unknown, although it has been associated with gallstones, high estrogen levels, cigarette smoking, alcohol, obesity, and the female gender. Approximately 70-90 percent of people with gallbladder cancer have gallstones. However, the opposite is not true,AS DR. HARI KRISHNA suggests, as most people with gallstones do not develop gallbladder cancers, and it is unclear how big of a risk gallstones pose. It has been suggested that infection with salmonella may also increase the risk of gallbladder cancers. Also, patients with inflammatory bowel disease (ulcerative colitis and Crohn's disease) are 10 times more likely to develop cancer of the extrahepatic biliary tract.
Q. What are the signs and symptoms of gallbladder cancer?
A. Unfortunately, there are no specific, surefire symptoms that suggest a diagnosis of gallbladder cancer. Usually, patients present with problems resulting from blockage of the bile ducts, such as jaundice, loss of appetite and weight loss. There may be a mass and/or pain in the abdomen, especially on the right under the ribcage. However, people often have no symptoms, or their symptoms closely mimic those of gallstones. The most common way gallbladder tumors are diagnosed is incidentally, during surgery performed to remove the gallbladder (cholecystectomy) for some other reason. About 1-2% cholecystectomies reveal a cancer of the gallbladder.
On patient examination, a healthcare provider (HCP) may detect jaundice of skin or the whites of the eyes, a mass in the right upper abdominal quadrant or around the belly button (periumbilical).
Q.How is gallbladder cancer diagnosed?
A. First and foremost, a HCP should always perform a thorough history and physical examination. Laboratory work should include metabolic chemistry and liver function panels to look for abnormal levels of various substances in the blood that are suggestive of general hepatobiliary disease. A urinalysis is usually done to evaluate urinary levels of some of these substances as well.
The role of tumor markers, [carbohydrate antigen 19-9 (CA 19-9), cancer antigen 125 (CA125), and carcinoembryonic antigen (CEA)] has not been established in gallbladder cancer.
Ultrasonography ( US) is the standard study done first in patients presenting with right upper quadrant pain. It allows HCPs to make a diagnosis of gallbladder cancer in about half of patients, and can also detect disease spread into the liver or bile ducts about half of the time. This is an important test, as it can help differentiate people who are having pain from gallstones from those who have gallbladder cancer. Endoscopic ultrasound, where a camera is inserted down through the mouth (while under sedation) allows the ultrasound probe to be placed closer to the gallbladder and appears to be more accurate than the traditional ultrasound which is placed against the abdominal wall. Endoscopic ultrasound may also better detect nodes and whether the tumor has spread beyond the gallbladder.
Computed tomography (CT) scans can also be helpful in patients with upper abdominal pain. They are better than US for detecting tumor invasion out of the gallbladder and disease spread to other sites in the abdomen or pelvis. About 70-80% of cases will have some degree of liver invasion, and so the combination of CT and US provides more accurate information.
Magnetic resonance imaging (MRI) has been useful in examining this region for disease spread into the liver or other tissues. This technology is particularly good for planning surgery, by evaluating surrounding blood vessels [magnetic resonance angiogram (MRA)] and bile duct passages [magnetic resonance cholangiogram (MRC)].
Cholangiography, either through the skin or the stomach, is a technique that allows HCPs to not only establish a diagnosis, but to locate the blockage and place a stent through the blockage to help alleviate the blockage.
Routine blood work may also help your physician determine whether there is a blockage in the bile ducts and how well the liver is functioning. There are tumor markers which can be tested for in the blood, such as CEA and CA 19-9. These markers can be elevated in cancer of the gallbladder, but are not good for diagnosis as they are non-specific and other conditions can cause them to be elevated.
Q. How is gallbladder cancer treated?
A. Cancer Healer is really effective in gall bladder cancer and brings marked improvement even at the last stages. It treats lung cancer promptly without any side effects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations
Q. What are the types of gallbladder cancer?
A. The majority of these cancers are "adenocarcinomas", with subtypes such as papillary, nodular, and tubular, depending on the appearance of the tumor cells under the microscope. Less common subtypes include: squamous cell, signet ring cell, and adenosquamous (adenoacanthoma).
Q. What causes gallbladder cancer? What are the risk factors?
A. The cause of gallbladder cancer is unknown, although it has been associated with gallstones, high estrogen levels, cigarette smoking, alcohol, obesity, and the female gender. Approximately 70-90 percent of people with gallbladder cancer have gallstones. However, the opposite is not true,AS DR. HARI KRISHNA suggests, as most people with gallstones do not develop gallbladder cancers, and it is unclear how big of a risk gallstones pose. It has been suggested that infection with salmonella may also increase the risk of gallbladder cancers. Also, patients with inflammatory bowel disease (ulcerative colitis and Crohn's disease) are 10 times more likely to develop cancer of the extrahepatic biliary tract.
Q. What are the signs and symptoms of gallbladder cancer?
A. Unfortunately, there are no specific, surefire symptoms that suggest a diagnosis of gallbladder cancer. Usually, patients present with problems resulting from blockage of the bile ducts, such as jaundice, loss of appetite and weight loss. There may be a mass and/or pain in the abdomen, especially on the right under the ribcage. However, people often have no symptoms, or their symptoms closely mimic those of gallstones. The most common way gallbladder tumors are diagnosed is incidentally, during surgery performed to remove the gallbladder (cholecystectomy) for some other reason. About 1-2% cholecystectomies reveal a cancer of the gallbladder.
On patient examination, a healthcare provider (HCP) may detect jaundice of skin or the whites of the eyes, a mass in the right upper abdominal quadrant or around the belly button (periumbilical).
Q.How is gallbladder cancer diagnosed?
A. First and foremost, a HCP should always perform a thorough history and physical examination. Laboratory work should include metabolic chemistry and liver function panels to look for abnormal levels of various substances in the blood that are suggestive of general hepatobiliary disease. A urinalysis is usually done to evaluate urinary levels of some of these substances as well.
The role of tumor markers, [carbohydrate antigen 19-9 (CA 19-9), cancer antigen 125 (CA125), and carcinoembryonic antigen (CEA)] has not been established in gallbladder cancer.
Ultrasonography ( US) is the standard study done first in patients presenting with right upper quadrant pain. It allows HCPs to make a diagnosis of gallbladder cancer in about half of patients, and can also detect disease spread into the liver or bile ducts about half of the time. This is an important test, as it can help differentiate people who are having pain from gallstones from those who have gallbladder cancer. Endoscopic ultrasound, where a camera is inserted down through the mouth (while under sedation) allows the ultrasound probe to be placed closer to the gallbladder and appears to be more accurate than the traditional ultrasound which is placed against the abdominal wall. Endoscopic ultrasound may also better detect nodes and whether the tumor has spread beyond the gallbladder.
Computed tomography (CT) scans can also be helpful in patients with upper abdominal pain. They are better than US for detecting tumor invasion out of the gallbladder and disease spread to other sites in the abdomen or pelvis. About 70-80% of cases will have some degree of liver invasion, and so the combination of CT and US provides more accurate information.
Magnetic resonance imaging (MRI) has been useful in examining this region for disease spread into the liver or other tissues. This technology is particularly good for planning surgery, by evaluating surrounding blood vessels [magnetic resonance angiogram (MRA)] and bile duct passages [magnetic resonance cholangiogram (MRC)].
Cholangiography, either through the skin or the stomach, is a technique that allows HCPs to not only establish a diagnosis, but to locate the blockage and place a stent through the blockage to help alleviate the blockage.
Routine blood work may also help your physician determine whether there is a blockage in the bile ducts and how well the liver is functioning. There are tumor markers which can be tested for in the blood, such as CEA and CA 19-9. These markers can be elevated in cancer of the gallbladder, but are not good for diagnosis as they are non-specific and other conditions can cause them to be elevated.
Q. How is gallbladder cancer treated?
A. Cancer Healer is really effective in gall bladder cancer and brings marked improvement even at the last stages. It treats lung cancer promptly without any side effects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations
PROSTATE CANCER
PROSTATE CANCER
Q. What is a prostate?
Q. What causes prostate cancer?
Q. Am I at risk for prostate cancer?
Q. What are the signs of prostate cancer?
Q. What screening tests are available?
Q. What are the treatments for prostate cancer?
Q. What is a prostate?
Ans. The prostate is a part of a reproductive organ which helps make and store seminal fluid. In adult men a typical prostate is about three centimeters long and weighs about twenty grams.It is located in the pelvis, under the urinary bladder and in front of the rectum. The prostate surrounds part of the urethra, the tube that carries urine from the bladder during urination and semen during ejaculation. Because of its location, prostate diseases often affect urination, ejaculation, and rarely defecation. The prostate contains many small glands which make about twenty percent of the fluid constituting semen.In prostate cancer the cells of these prostate glands mutate into cancer cells. The prostate glands require male hormones, known as androgens, to work properly. Androgens include testosterone, which is made in the testes; dehydroepiandrosterone, made in the adrenal glands; and dihydrotestosterone, which is converted from testosterone within the prostate itself. Androgens are also responsible for secondary sex characteristics such as facial hair and increased muscle mass.
Q. What causes prostate cancer?
A. The cause of prostate cancer is unknown, but the cancer is thought not to be related to benign prostatic hypertrophy (BPH). The risk (predisposing) factors for prostate cancer include advancing age, genetics (heredity), hormonal influences, and such environmental factors as toxins, chemicals, and industrial products. The chances of developing prostate cancer increase with age. Thus, prostate cancer under age 40 is extremely rare, while it is common in men older than 80 years of age. As a matter of fact, some studies have suggested that among men over 80, between 50 and 80% of them may have prostate cancer!
Genetics (heredity), as just mentioned, plays a role in the risk of developing a prostate cancer. Environment, diet, and other unknown factors, however, can modify such genetic predispositions. For example, prostate cancer is uncommon in Japanese men living in their native Japan. However, when these men move to the United States, their incidence of prostate cancer rises significantly. Prostate cancer is also more common among family members of individuals with prostate cancer. Thus, a person whose father, grandfather, or even uncle has prostate cancer is at an increased risk for also developing prostate cancer. To date, however, no specific prostate cancer gene has been identified and verified. (Genes, which are situated on chromosomes within the nucleus of cells, are the chemical compounds that determine specific traits in individuals.)Testosterone, the male hormone, directly stimulates the growth of both normal prostate tissue and prostate cancer cells. Not surprisingly, therefore, this hormone is thought to be involved in the development and growth of prostate cancer. The important implication of the role of this hormone is that decreasing the level of testosterone should be (and usually is) effective in inhibiting the growth of prostate cancer.Environmental factors, such as cigarette smoking and diet that are high in saturated fat, seem to increase the risk of prostate cancer. Additional substances or toxins in the environment or from industrial sources might also promote the development of prostate cancer, but these have not yet been clearly identified.
Q. Am I at risk for prostate cancer?
A. Every man over the age of 45 is at risk for prostate cancer. Although prostate cancer can occasionally strike younger men, the risk of getting prostate cancer increases with age and more than 70% of men diagnosed with prostate cancer are over the age of 65. Prostate cancer is the most common cancer that men get .Although there are several known risk factors for getting prostate cancer, no one knows exactly why one man gets it and another doesn't. Some of the most important risk factors for prostate cancer include age, ethnicity, genetics and diet. Age is generally considered the most important risk factor for prostate cancer, as elaborated by Dr. Tarang Krishna. The incidence of prostate cancer rises quickly after the age of 60, and the majority of men will have some form of prostate cancer after the age of 80. One of the sayings about prostate cancer is that older men (over the age of 80) die with prostate cancer not from prostate cancer. This saying means that many older men have microscopic disease that doesn't shorten their life expectancy because the cancer takes a long time to grow and become clinically important. However, this saying is only a generalization; sometimes prostate cancer can grow quickly even in older patients.
Another important risk factor for prostate cancer is ethnicity. No one knows exactly why, but prostate cancer is more common in African-American and Latino men than Caucasian men. African-American men have a 1.6 fold higher chance of getting and dying from prostate cancer than Caucasian men. Asian and Native American men have the lowest chances of getting prostate cancer. Some doctors believe that genetic differences are important in explaining the different rates of prostate cancer between different ethnic groups; however, there is some evidence that differences in diets may be the cause. When Asian men move to Western countries like the United States, their chances of getting prostate cancer rise. Men who live in the United States and Northern Europe have the highest rates of prostate cancer, while men who live in South America, Central America, Africa, and Asia all have much lower chances of developing prostate cancer.
There is some evidence that a man's diet may affect his risk of developing prostate cancer. The most common dietary culprit implicated in raising prostate cancer risk is a high fat diet, particularly a diet high in animal fats. Also, a few studies have suggested that a diet low in vegetables causes an increased risk of prostate cancer. There are a few foods that have been implicated in decreasing prostate cancer risk: a diet high in tomatoes (lycopene) has been suggested as well as diet high in omega-3-fatty acids (oils found in fish like salmon and mackerel).
A family history of prostate cancer increases a man's chances of developing the disease, as per Dr. Hari Krishna.Cancer-Healer works marvellously in cases of heredity prostate cancers in men. This increase shows itself when a man has either a father or brothers (or both) with prostate cancer, and is even greater when his relatives develop prostate cancer at a young age. A variety of different genetic factors are currently being researched. Variations and mutations in certain genes may be responsible for some increases in prostate cancer rates in families. Men who carry mutations in genes known as BRCA1 or BRCA2 (these are genes implicated in breast and ovarian cancer in women) may have a 2 to 5 fold increase in prostate cancer risk.
Q. What are the signs of prostate cancer?
A. Most early prostate cancers are detected with PSA tests or digital rectal exams before they cause any symptoms. However, more advanced prostate cancers can cause a variety of symptoms including:
1- trouble starting urination
2- urinating much more frequently than usual
3- the feeling that you can't release all of your urine
4- pain on urination or ejaculation
5- blood in your urine or semen
6- impotence
7- bone pain
All of these symptoms can be caused by a variety of things besides prostate cancer, so experiencing them doesn't necessarily mean you have prostate cancer. As per Dr. Hari Krishna, When older men have problems urinating, it is usually caused by process called benign prostatic hyperplasia (BPH) which is not prostate cancer. If you experience any of these symptoms, you need to see your doctor for evaluation.
Q. What screening tests are available?
A. We know that prostate cancer usually grows very slowly, so intuitively it would make sense that we could reduce mortality from prostate cancer by picking it up early so it could be treated before it spreads. However, in order for a screening test to be fully embraced, we need to prove that picking up a disease early actually does help reduce the number of deaths. Right now, there is no good data showing that screening for prostate cancer reduces deaths from prostate cancer.
Currently, there are two methods that physicians use to screen for prostate cancer. One of them is called a digital rectal exam (DRE). A digital rectal exam is done in your primary care physician's office. Because your prostate is so close to your rectum, your doctor can feel it by inserting a gloved, lubricated finger into your anus. Your doctor can feel if there are lumps, asymmetries, or if your prostate is enlarged. A digital rectal exam is uncomfortable, but not painful. It is a useful test, but it is not perfect because some small cancers can be missed and only the bottom and sides of the prostate can be examined in this manner. Although it isn't a perfect test, it becomes more useful when it is combined with another test called a PSA.
A PSA (prostate specific antigen) test is a blood test that looks for a protein that the prostate makes. Normal prostate tissue makes a little bit of PSA, but prostate cancer usually makes much more. By checking to see if your PSA is elevated, your doctor can screen you for prostate cancer. The PSA test isn't perfect either, because some tumors won't elevate the PSA and some other processes (like benign prostatic hyperplasia and prostatitis) can cause it to be falsely elevated. However, the higher your PSA is, the more likely the elevation is caused by a prostate cancer. The cut-off that your doctor usually uses is 4.0 ng/ml, meaning that anything below 4.0 ng/ml is normal and anything above it is abnormal. If your PSA is elevated, or you have an abnormal digital rectal exam, then you need to get further evaluation; however, this doesn't necessarily mean that you have prostate cancer. The only way to know for sure whether or not you have cancer is to get a sample of your prostate from a biopsy.
Q. What are the treatments for prostate cancer?
A. There are many different ways to treat prostate cancer, and you will most likely be consulting multiple types of doctors before making a final decision. Physicians are not always in agreement as to the way to proceed because there haven't been enough large trials that compare the different treatment modalities. For prostate cancer, it is important that you get a second opinion and you should talk to both urologists and radiation oncologists to hear about the benefits and risks of surgery, hormonal therapy and radiation in your particular case. If your prostate cancer has already spread at the time of diagnosis, you will also need a medical oncologist to talk about chemotherapy. The most important thing is to discuss your options and make a decision that suits your lifestyle, beliefs and values.
CANCER-HEALER is a treatment where the response is seen in the PSA levels, with the improvement seen in generally all men with prostate cancers. The patients who even opt for chemotherapy also see marked improvement who take CANCER-HEALER in parallel with chemotherapy. Cancer-Healer is effective in prostate cancers.
Q. What is a prostate?
Q. What causes prostate cancer?
Q. Am I at risk for prostate cancer?
Q. What are the signs of prostate cancer?
Q. What screening tests are available?
Q. What are the treatments for prostate cancer?
Q. What is a prostate?
Ans. The prostate is a part of a reproductive organ which helps make and store seminal fluid. In adult men a typical prostate is about three centimeters long and weighs about twenty grams.It is located in the pelvis, under the urinary bladder and in front of the rectum. The prostate surrounds part of the urethra, the tube that carries urine from the bladder during urination and semen during ejaculation. Because of its location, prostate diseases often affect urination, ejaculation, and rarely defecation. The prostate contains many small glands which make about twenty percent of the fluid constituting semen.In prostate cancer the cells of these prostate glands mutate into cancer cells. The prostate glands require male hormones, known as androgens, to work properly. Androgens include testosterone, which is made in the testes; dehydroepiandrosterone, made in the adrenal glands; and dihydrotestosterone, which is converted from testosterone within the prostate itself. Androgens are also responsible for secondary sex characteristics such as facial hair and increased muscle mass.
Q. What causes prostate cancer?
A. The cause of prostate cancer is unknown, but the cancer is thought not to be related to benign prostatic hypertrophy (BPH). The risk (predisposing) factors for prostate cancer include advancing age, genetics (heredity), hormonal influences, and such environmental factors as toxins, chemicals, and industrial products. The chances of developing prostate cancer increase with age. Thus, prostate cancer under age 40 is extremely rare, while it is common in men older than 80 years of age. As a matter of fact, some studies have suggested that among men over 80, between 50 and 80% of them may have prostate cancer!
Genetics (heredity), as just mentioned, plays a role in the risk of developing a prostate cancer. Environment, diet, and other unknown factors, however, can modify such genetic predispositions. For example, prostate cancer is uncommon in Japanese men living in their native Japan. However, when these men move to the United States, their incidence of prostate cancer rises significantly. Prostate cancer is also more common among family members of individuals with prostate cancer. Thus, a person whose father, grandfather, or even uncle has prostate cancer is at an increased risk for also developing prostate cancer. To date, however, no specific prostate cancer gene has been identified and verified. (Genes, which are situated on chromosomes within the nucleus of cells, are the chemical compounds that determine specific traits in individuals.)Testosterone, the male hormone, directly stimulates the growth of both normal prostate tissue and prostate cancer cells. Not surprisingly, therefore, this hormone is thought to be involved in the development and growth of prostate cancer. The important implication of the role of this hormone is that decreasing the level of testosterone should be (and usually is) effective in inhibiting the growth of prostate cancer.Environmental factors, such as cigarette smoking and diet that are high in saturated fat, seem to increase the risk of prostate cancer. Additional substances or toxins in the environment or from industrial sources might also promote the development of prostate cancer, but these have not yet been clearly identified.
Q. Am I at risk for prostate cancer?
A. Every man over the age of 45 is at risk for prostate cancer. Although prostate cancer can occasionally strike younger men, the risk of getting prostate cancer increases with age and more than 70% of men diagnosed with prostate cancer are over the age of 65. Prostate cancer is the most common cancer that men get .Although there are several known risk factors for getting prostate cancer, no one knows exactly why one man gets it and another doesn't. Some of the most important risk factors for prostate cancer include age, ethnicity, genetics and diet. Age is generally considered the most important risk factor for prostate cancer, as elaborated by Dr. Tarang Krishna. The incidence of prostate cancer rises quickly after the age of 60, and the majority of men will have some form of prostate cancer after the age of 80. One of the sayings about prostate cancer is that older men (over the age of 80) die with prostate cancer not from prostate cancer. This saying means that many older men have microscopic disease that doesn't shorten their life expectancy because the cancer takes a long time to grow and become clinically important. However, this saying is only a generalization; sometimes prostate cancer can grow quickly even in older patients.
Another important risk factor for prostate cancer is ethnicity. No one knows exactly why, but prostate cancer is more common in African-American and Latino men than Caucasian men. African-American men have a 1.6 fold higher chance of getting and dying from prostate cancer than Caucasian men. Asian and Native American men have the lowest chances of getting prostate cancer. Some doctors believe that genetic differences are important in explaining the different rates of prostate cancer between different ethnic groups; however, there is some evidence that differences in diets may be the cause. When Asian men move to Western countries like the United States, their chances of getting prostate cancer rise. Men who live in the United States and Northern Europe have the highest rates of prostate cancer, while men who live in South America, Central America, Africa, and Asia all have much lower chances of developing prostate cancer.
There is some evidence that a man's diet may affect his risk of developing prostate cancer. The most common dietary culprit implicated in raising prostate cancer risk is a high fat diet, particularly a diet high in animal fats. Also, a few studies have suggested that a diet low in vegetables causes an increased risk of prostate cancer. There are a few foods that have been implicated in decreasing prostate cancer risk: a diet high in tomatoes (lycopene) has been suggested as well as diet high in omega-3-fatty acids (oils found in fish like salmon and mackerel).
A family history of prostate cancer increases a man's chances of developing the disease, as per Dr. Hari Krishna.Cancer-Healer works marvellously in cases of heredity prostate cancers in men. This increase shows itself when a man has either a father or brothers (or both) with prostate cancer, and is even greater when his relatives develop prostate cancer at a young age. A variety of different genetic factors are currently being researched. Variations and mutations in certain genes may be responsible for some increases in prostate cancer rates in families. Men who carry mutations in genes known as BRCA1 or BRCA2 (these are genes implicated in breast and ovarian cancer in women) may have a 2 to 5 fold increase in prostate cancer risk.
Q. What are the signs of prostate cancer?
A. Most early prostate cancers are detected with PSA tests or digital rectal exams before they cause any symptoms. However, more advanced prostate cancers can cause a variety of symptoms including:
1- trouble starting urination
2- urinating much more frequently than usual
3- the feeling that you can't release all of your urine
4- pain on urination or ejaculation
5- blood in your urine or semen
6- impotence
7- bone pain
All of these symptoms can be caused by a variety of things besides prostate cancer, so experiencing them doesn't necessarily mean you have prostate cancer. As per Dr. Hari Krishna, When older men have problems urinating, it is usually caused by process called benign prostatic hyperplasia (BPH) which is not prostate cancer. If you experience any of these symptoms, you need to see your doctor for evaluation.
Q. What screening tests are available?
A. We know that prostate cancer usually grows very slowly, so intuitively it would make sense that we could reduce mortality from prostate cancer by picking it up early so it could be treated before it spreads. However, in order for a screening test to be fully embraced, we need to prove that picking up a disease early actually does help reduce the number of deaths. Right now, there is no good data showing that screening for prostate cancer reduces deaths from prostate cancer.
Currently, there are two methods that physicians use to screen for prostate cancer. One of them is called a digital rectal exam (DRE). A digital rectal exam is done in your primary care physician's office. Because your prostate is so close to your rectum, your doctor can feel it by inserting a gloved, lubricated finger into your anus. Your doctor can feel if there are lumps, asymmetries, or if your prostate is enlarged. A digital rectal exam is uncomfortable, but not painful. It is a useful test, but it is not perfect because some small cancers can be missed and only the bottom and sides of the prostate can be examined in this manner. Although it isn't a perfect test, it becomes more useful when it is combined with another test called a PSA.
A PSA (prostate specific antigen) test is a blood test that looks for a protein that the prostate makes. Normal prostate tissue makes a little bit of PSA, but prostate cancer usually makes much more. By checking to see if your PSA is elevated, your doctor can screen you for prostate cancer. The PSA test isn't perfect either, because some tumors won't elevate the PSA and some other processes (like benign prostatic hyperplasia and prostatitis) can cause it to be falsely elevated. However, the higher your PSA is, the more likely the elevation is caused by a prostate cancer. The cut-off that your doctor usually uses is 4.0 ng/ml, meaning that anything below 4.0 ng/ml is normal and anything above it is abnormal. If your PSA is elevated, or you have an abnormal digital rectal exam, then you need to get further evaluation; however, this doesn't necessarily mean that you have prostate cancer. The only way to know for sure whether or not you have cancer is to get a sample of your prostate from a biopsy.
Q. What are the treatments for prostate cancer?
A. There are many different ways to treat prostate cancer, and you will most likely be consulting multiple types of doctors before making a final decision. Physicians are not always in agreement as to the way to proceed because there haven't been enough large trials that compare the different treatment modalities. For prostate cancer, it is important that you get a second opinion and you should talk to both urologists and radiation oncologists to hear about the benefits and risks of surgery, hormonal therapy and radiation in your particular case. If your prostate cancer has already spread at the time of diagnosis, you will also need a medical oncologist to talk about chemotherapy. The most important thing is to discuss your options and make a decision that suits your lifestyle, beliefs and values.
CANCER-HEALER is a treatment where the response is seen in the PSA levels, with the improvement seen in generally all men with prostate cancers. The patients who even opt for chemotherapy also see marked improvement who take CANCER-HEALER in parallel with chemotherapy. Cancer-Healer is effective in prostate cancers.
STOMACH CANCER
STOMACH CANCER
Q. What is stomach cancer?
Q. How common is stomach cancer?
Q. How is stomach cancer diagnosed?
Q. What are the symptoms of stomach cancer?
Q. Does stomach cancer run in families?
Q. Who is at risk of stomach cancer?
Q. What causes stomach cancer?
Q. Who is most likely to get stomach cancer?
Q. How effective are the treatments?
Q. What is stomach cancer?
A. There are two main types of stomach cancer called the intestinal and diffuse types. Intestinal stomach cancer is more common amongst older patients and patients from high-risk groups (eg the Japanese and Koreans). Diffuse stomach cancer is found more frequently in women and people with blood group A. This variety of stomach cancer is more difficult to treat.
Q. How common is stomach cancer?
A. Stomach cancer is more common than most people realise. Each year there are many cases where the diet or the H. pylori leads to the stomach cancers worldwide.
Q. How is stomach cancer diagnosed?
A. Normally, a procedure called endoscopy is used to see the inside of the stomach. Under sedation, a thin fibre-optic tube is passed down the throat. The inside of the stomach is lit up and photographed. Any growths can be spotted relatively easily by this method. Sometimes, more sophisticated endoscopes are used which can take a small sample of the stomach lining or even take an ultrasound scan from the inside of the stomach. In some cases, a barium meal is used instead of endoscopy. The patient is given a white liquid to drink, containing the element barium. Then the stomach is observed through an X-ray screen. The way that the barium flows through the stomach will reveal any growths.
Q. What are the symptoms of stomach cancer?
A. Early stomach cancer often has no symptoms or merely causes a stomach ache. As it becomes more advanced, it can cause loss of appetite, nausea, vomiting, severe stomach pain and weight loss. Since many of these symptoms are also caused by food poisoning, stomach ulcers and several other conditions, all too often stomach cancers are not diagnosed until they are quite advanced.
Q. Does stomach cancer run in families?
A. A few cases of stomach cancer (about one in ten) appear to run in the family. We do not yet understand which genes are involved, so genetic testing is not yet possible.
Q. Who is at risk of stomach cancer?
A. Diet - eating a diet high in preserved foods has been linked with higher rates of stomach cancer (see above). Gender - stomach cancer is almost twice as common in men as in women. Other diseases - people with pernicious anaemia or achlorhydria are more likely to get stomach cancer. Infections- stomach infections by a bacterium called Helicobacter pylori, which is a major cause of stomach ulcers, is also associated with a much higher risk of stomach cancer. Smoking - this is also known to increase the risk of getting stomach cancer.
Q. What causes stomach cancer?
A. The function of the stomach and the wide differences in stomach cancer rates between different countries make it almost certain that the food we eat is - or was - a major factor in the cause. The likely reason that the number of cases has dropped over the last fifty years is that refrigeration of food became common and people ate less pickled, salted, smoked and cured foods.
Q. Who is most likely to get stomach cancer?
A. Like many cancers, this type is most common in older people. Few cases occur below 50 years of age and the highest rates are in men and women over 70. Men are twice as likely to get stomach cancer as women. The risk of stomach cancer also depends a lot on where you live (see above). Korea and Japan have the highest rates, ten times the rate in the USA.
Q. How effective are the treatments?
A. Cancer Healer is really effective in Stomach Cancer and brings marked improvement even at the last stages. It treats stomach cancer promptly without any sideeffects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations.
Q. What is stomach cancer?
Q. How common is stomach cancer?
Q. How is stomach cancer diagnosed?
Q. What are the symptoms of stomach cancer?
Q. Does stomach cancer run in families?
Q. Who is at risk of stomach cancer?
Q. What causes stomach cancer?
Q. Who is most likely to get stomach cancer?
Q. How effective are the treatments?
Q. What is stomach cancer?
A. There are two main types of stomach cancer called the intestinal and diffuse types. Intestinal stomach cancer is more common amongst older patients and patients from high-risk groups (eg the Japanese and Koreans). Diffuse stomach cancer is found more frequently in women and people with blood group A. This variety of stomach cancer is more difficult to treat.
Q. How common is stomach cancer?
A. Stomach cancer is more common than most people realise. Each year there are many cases where the diet or the H. pylori leads to the stomach cancers worldwide.
Q. How is stomach cancer diagnosed?
A. Normally, a procedure called endoscopy is used to see the inside of the stomach. Under sedation, a thin fibre-optic tube is passed down the throat. The inside of the stomach is lit up and photographed. Any growths can be spotted relatively easily by this method. Sometimes, more sophisticated endoscopes are used which can take a small sample of the stomach lining or even take an ultrasound scan from the inside of the stomach. In some cases, a barium meal is used instead of endoscopy. The patient is given a white liquid to drink, containing the element barium. Then the stomach is observed through an X-ray screen. The way that the barium flows through the stomach will reveal any growths.
Q. What are the symptoms of stomach cancer?
A. Early stomach cancer often has no symptoms or merely causes a stomach ache. As it becomes more advanced, it can cause loss of appetite, nausea, vomiting, severe stomach pain and weight loss. Since many of these symptoms are also caused by food poisoning, stomach ulcers and several other conditions, all too often stomach cancers are not diagnosed until they are quite advanced.
Q. Does stomach cancer run in families?
A. A few cases of stomach cancer (about one in ten) appear to run in the family. We do not yet understand which genes are involved, so genetic testing is not yet possible.
Q. Who is at risk of stomach cancer?
A. Diet - eating a diet high in preserved foods has been linked with higher rates of stomach cancer (see above). Gender - stomach cancer is almost twice as common in men as in women. Other diseases - people with pernicious anaemia or achlorhydria are more likely to get stomach cancer. Infections- stomach infections by a bacterium called Helicobacter pylori, which is a major cause of stomach ulcers, is also associated with a much higher risk of stomach cancer. Smoking - this is also known to increase the risk of getting stomach cancer.
Q. What causes stomach cancer?
A. The function of the stomach and the wide differences in stomach cancer rates between different countries make it almost certain that the food we eat is - or was - a major factor in the cause. The likely reason that the number of cases has dropped over the last fifty years is that refrigeration of food became common and people ate less pickled, salted, smoked and cured foods.
Q. Who is most likely to get stomach cancer?
A. Like many cancers, this type is most common in older people. Few cases occur below 50 years of age and the highest rates are in men and women over 70. Men are twice as likely to get stomach cancer as women. The risk of stomach cancer also depends a lot on where you live (see above). Korea and Japan have the highest rates, ten times the rate in the USA.
Q. How effective are the treatments?
A. Cancer Healer is really effective in Stomach Cancer and brings marked improvement even at the last stages. It treats stomach cancer promptly without any sideeffects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations.
OVARIAN CANCER
OVARIAN CANCER
Q. Where are the ovaries?
Q. What are the risk factors for ovarian cancer?
Q. How dangerous are ovarian cancers?
Q. How common is ovarian cancer?
Q. Can we screen for ovarian cancer?
Q. Does ovarian cancer run in families?
Q. Does the diet affect the risk of ovarian cancer?
Q. How is ovarian cancer treated?
Q. How is ovarian cancer diagnosed?
Q. What are the symptoms of ovarian cancer?
Q. How effective are the treatments?
Q. Where are the ovaries?
A. The ovaries are above the womb and connected to it by two short tubes (the Fallopian tubes). This means that they are below and to either side of the navel.
Q. What are the risk factors for ovarian cancer?
A. Like most cancers it is more common with increasing age. The other risk factor is if you carry certain genes (see below). Having children reduces the risk: women with three or four children have only half the risk of a childless woman. Infertile women (ie women who cannot conceive despite trying for several years) appear to have an even higher risk than other childless women. Taking the contraceptive pill reduces the risk of ovarian cancer by somewhere between a third and a half, depending on how long it is taken for. Although the effect of hormone replacement therapy (HRT) on ovarian cancer risk has been studied, the results are unclear. Some studies have found an increased risk, but analysis of all the published research shows conflicting results. There have been some reports claiming that using talc in the genital area increases the risk of ovarian cancer. However, most of the research conducted on this has not produced reliable findings and there is no good evidence to support these claims.
Q. How dangerous are ovarian cancers?
A. Most of the women died of ovarian cancer, making it a more common cause of death than cervical and uterine cancer combined. The ovarian cancers can prove to be very fatal by ill chosen modes of treatment or if overlooked or neglected. The alarming levels of Ca-125 can also confirm the same.
Q. How common is ovarian cancer?
A. Ovarian Cancers are common with the family history showing the same or even in the women showing histories of major operations or use of contraceptive pills,as per the data which reveals the same
Q. Can we screen for ovarian cancer?
A. There is no reliable method of screening for ovarian cancer. However, both the CA125 blood test and vaginal ultrasound are currently being tested as possible methods for screening women for ovarian cancer.
Q. Does ovarian cancer run in families?
A. There are several genes, which are known to carry increased risks of various cancers, which can run in families. The BRCA1 and BRCA2 genes were originally discovered because they cause an increased risk of breast cancer, but we now know that they also substantially increase the risk of ovarian cancer. The HNPCC gene was discovered because it increases the risk of colon cancer, but women with this gene also have a greater chance of getting ovarian cancer. Overall, if you have one close relative (mother, sister or daughter) who has had ovarian cancer, your risk goes up about 4-fold. If you have two cases amongst close relatives, your risk goes up 10-fold or more.
Q. Does the diet affect the risk of ovarian cancer?
A. There is some evidence that being overweight can increase your risk of ovarian cancer. Some research has suggested that beta-carotene in the diet can reduce the risk of this cancer, although this finding has not yet been confirmed.
Q. How is ovarian cancer treated?
A. Cancer Healer is really effective in Ovary Cancer and brings marked improvement even at the last stages. It treats ovarian cancer promptly without any side effects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations
Q. How is ovarian cancer diagnosed?
A. If ovarian cancer is suspected, two main tests are used to make the diagnosis. First, an ultrasound scan of the abdomen is performed. Sometimes the scan is taken from inside the vagina. The second test is to measure the level of the CA125 marker in the blood. Neither of these tests gives a definite diagnosis of ovarian cancer, but if both tests are positive, the patient is usually referred to a surgeon who will operate to see if the ovaries show any signs of cancer.
Q. What are the symptoms of ovarian cancer?
A. There are few clear symptoms of ovarian cancer. Typically it can cause pain in the abdomen, a feeling of being bloated, fatigue, weight loss, or problems with urination. However, these can all be caused by a number of other diseases. This makes it difficult to diagnose ovarian cancer by symptoms alone.
Q. How effective are the treatments?
A. 4 out of every 5 women with ovarian cancer can be cured by Cancer Healer. Like all other cancers, the stage at which ovarian cancer is diagnosed determines how easily it is to cure.If diagnosed and treated while the cancer is confined to the ovaries nearly 80% of the women can be cured, mentions Dr. Tarang.
Q. Where are the ovaries?
Q. What are the risk factors for ovarian cancer?
Q. How dangerous are ovarian cancers?
Q. How common is ovarian cancer?
Q. Can we screen for ovarian cancer?
Q. Does ovarian cancer run in families?
Q. Does the diet affect the risk of ovarian cancer?
Q. How is ovarian cancer treated?
Q. How is ovarian cancer diagnosed?
Q. What are the symptoms of ovarian cancer?
Q. How effective are the treatments?
Q. Where are the ovaries?
A. The ovaries are above the womb and connected to it by two short tubes (the Fallopian tubes). This means that they are below and to either side of the navel.
Q. What are the risk factors for ovarian cancer?
A. Like most cancers it is more common with increasing age. The other risk factor is if you carry certain genes (see below). Having children reduces the risk: women with three or four children have only half the risk of a childless woman. Infertile women (ie women who cannot conceive despite trying for several years) appear to have an even higher risk than other childless women. Taking the contraceptive pill reduces the risk of ovarian cancer by somewhere between a third and a half, depending on how long it is taken for. Although the effect of hormone replacement therapy (HRT) on ovarian cancer risk has been studied, the results are unclear. Some studies have found an increased risk, but analysis of all the published research shows conflicting results. There have been some reports claiming that using talc in the genital area increases the risk of ovarian cancer. However, most of the research conducted on this has not produced reliable findings and there is no good evidence to support these claims.
Q. How dangerous are ovarian cancers?
A. Most of the women died of ovarian cancer, making it a more common cause of death than cervical and uterine cancer combined. The ovarian cancers can prove to be very fatal by ill chosen modes of treatment or if overlooked or neglected. The alarming levels of Ca-125 can also confirm the same.
Q. How common is ovarian cancer?
A. Ovarian Cancers are common with the family history showing the same or even in the women showing histories of major operations or use of contraceptive pills,as per the data which reveals the same
Q. Can we screen for ovarian cancer?
A. There is no reliable method of screening for ovarian cancer. However, both the CA125 blood test and vaginal ultrasound are currently being tested as possible methods for screening women for ovarian cancer.
Q. Does ovarian cancer run in families?
A. There are several genes, which are known to carry increased risks of various cancers, which can run in families. The BRCA1 and BRCA2 genes were originally discovered because they cause an increased risk of breast cancer, but we now know that they also substantially increase the risk of ovarian cancer. The HNPCC gene was discovered because it increases the risk of colon cancer, but women with this gene also have a greater chance of getting ovarian cancer. Overall, if you have one close relative (mother, sister or daughter) who has had ovarian cancer, your risk goes up about 4-fold. If you have two cases amongst close relatives, your risk goes up 10-fold or more.
Q. Does the diet affect the risk of ovarian cancer?
A. There is some evidence that being overweight can increase your risk of ovarian cancer. Some research has suggested that beta-carotene in the diet can reduce the risk of this cancer, although this finding has not yet been confirmed.
Q. How is ovarian cancer treated?
A. Cancer Healer is really effective in Ovary Cancer and brings marked improvement even at the last stages. It treats ovarian cancer promptly without any side effects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations
Q. How is ovarian cancer diagnosed?
A. If ovarian cancer is suspected, two main tests are used to make the diagnosis. First, an ultrasound scan of the abdomen is performed. Sometimes the scan is taken from inside the vagina. The second test is to measure the level of the CA125 marker in the blood. Neither of these tests gives a definite diagnosis of ovarian cancer, but if both tests are positive, the patient is usually referred to a surgeon who will operate to see if the ovaries show any signs of cancer.
Q. What are the symptoms of ovarian cancer?
A. There are few clear symptoms of ovarian cancer. Typically it can cause pain in the abdomen, a feeling of being bloated, fatigue, weight loss, or problems with urination. However, these can all be caused by a number of other diseases. This makes it difficult to diagnose ovarian cancer by symptoms alone.
Q. How effective are the treatments?
A. 4 out of every 5 women with ovarian cancer can be cured by Cancer Healer. Like all other cancers, the stage at which ovarian cancer is diagnosed determines how easily it is to cure.If diagnosed and treated while the cancer is confined to the ovaries nearly 80% of the women can be cured, mentions Dr. Tarang.
MALIGNANT MESIOTHELIOMA
MALIGNANT MESIOTHELIOMA
Q. Am I at risk for mesothelioma?
Q. What are the signs of mesothelioma?
Q. How is mesothelioma diagnosed and staged?
Q. Am I at risk for mesothelioma?
A. Mesothelioma is a rare cancer. It is more common in men, which is due in most part to work-related exposure to asbestos. Risk also increases with age. The biggest risk factor for developing the disease is exposure to asbestos, accounting for 70 to 80 percent of all cases. Asbestos has been used in many products, including cement, brake linings, roof shingles, flooring products, textiles, and insulation. Particles can be released from these products, particularly during the manufacturing process, and inhaled. Prior to knowing the dangers, asbestos miners and other workers exposed to asbestos worked without wearing any protection. Smoking does not seem to increase the risk of developing the disease.
Q. What are the signs of mesothelioma?
A. The symptoms of mesothelioma are caused by a build-up of tumor tissue surrounding the lung and fluid in the pleural space that prevents the lung from expanding fully. This causes pressure on the lung, leading to pain and shortness of breath. As the disease progresses, patients may lose weight and have a dry, hacking cough. In the abdomen, this fluid and tumor tissue causes abdominal swelling, pain and weight loss.
Q. How is mesothelioma diagnosed and staged?
A. Patients who present with symptoms worrisome for mesothelioma may have a chest x-ray done, indicating a build-up of fluid in the lining of the lung. These patients would then undergo CT scan to further evaluate the cancer. In the case of abdominal mesothelioma, a CT scan . Advance tests include a PET-CT SCAN which would indicate the presence of active disease. Patients would then undergo a biopsy to have the diagnosis confirmed. In the lung, a thoracoscope is used to go through the chest wall, between the ribs to obtain a sample of the tissue. A peritoneoscope is used to enter the abdomen to obtain a tissue sample in abdominal mesothelioma. Staging refers to determining the extent of the disease and this dictates the treatment. Physicians use the TNM system (also called tumor - node - metastasis system). This describes the size of the tumor (T), if the lymph nodes are involved (N), and if it has spread to other areas of the body (M). This is then interpreted to a stage between one and four. Patients with earlier stage tumors tend to live longer and respond better to available treatments.
Q. Am I at risk for mesothelioma?
Q. What are the signs of mesothelioma?
Q. How is mesothelioma diagnosed and staged?
Q. Am I at risk for mesothelioma?
A. Mesothelioma is a rare cancer. It is more common in men, which is due in most part to work-related exposure to asbestos. Risk also increases with age. The biggest risk factor for developing the disease is exposure to asbestos, accounting for 70 to 80 percent of all cases. Asbestos has been used in many products, including cement, brake linings, roof shingles, flooring products, textiles, and insulation. Particles can be released from these products, particularly during the manufacturing process, and inhaled. Prior to knowing the dangers, asbestos miners and other workers exposed to asbestos worked without wearing any protection. Smoking does not seem to increase the risk of developing the disease.
Q. What are the signs of mesothelioma?
A. The symptoms of mesothelioma are caused by a build-up of tumor tissue surrounding the lung and fluid in the pleural space that prevents the lung from expanding fully. This causes pressure on the lung, leading to pain and shortness of breath. As the disease progresses, patients may lose weight and have a dry, hacking cough. In the abdomen, this fluid and tumor tissue causes abdominal swelling, pain and weight loss.
Q. How is mesothelioma diagnosed and staged?
A. Patients who present with symptoms worrisome for mesothelioma may have a chest x-ray done, indicating a build-up of fluid in the lining of the lung. These patients would then undergo CT scan to further evaluate the cancer. In the case of abdominal mesothelioma, a CT scan . Advance tests include a PET-CT SCAN which would indicate the presence of active disease. Patients would then undergo a biopsy to have the diagnosis confirmed. In the lung, a thoracoscope is used to go through the chest wall, between the ribs to obtain a sample of the tissue. A peritoneoscope is used to enter the abdomen to obtain a tissue sample in abdominal mesothelioma. Staging refers to determining the extent of the disease and this dictates the treatment. Physicians use the TNM system (also called tumor - node - metastasis system). This describes the size of the tumor (T), if the lymph nodes are involved (N), and if it has spread to other areas of the body (M). This is then interpreted to a stage between one and four. Patients with earlier stage tumors tend to live longer and respond better to available treatments.
LUNG CANCER
LUNG CANCER
Q. Does diet affect the risk of getting lung cancer?
Q. Are there different types of lung cancer?
Q. What causes lung cancer?
Q. Does lung cancer run in families?
Q. What are the symptoms of lung cancer?
Q. How is lung cancer treated?
Q. How is lung cancer diagnosed?
Q. Does diet affect the risk of getting lung cancer?
A. This is still being investigated, but research to date has not found any link between diet and lung cancer.
Q. Are there different types of lung cancer?
A. There are four main types of lung cancer: small cell lung cancer, squamous cell carcinoma, large cell carcinoma and adenocarcinoma Tobacco smoking is strongly linked to the first three but only weakly linked to adenocarcinoma However, this type of lung cancer has been linked to the use of low-tar cigarettes.
Q. What causes lung cancer?
A. The vast majority - over 80% - of lung cancers are caused by smoking tobacco or by indirect exposure to tobacco smoke (passive smoking). The other main causes are breathing industrial chemicals such as asbestos, arsenic and polycyclic hydrocarbons or the natural radioactive gas, radon.
Q. Does lung cancer run in families?
A. There are very few, if any, inherited conditions that increase the risk of lung cancer in non-smokers. However, not all of the people who smoke get lung cancer and there may be an inherited component which influences whether or not smoking will cause lung cancer.
Q. What are the symptoms of lung cancer?
A. There are a variety of symptoms of lung cancer, including difficulty breathing, coughing up blood, chest pain, loss of appetite, weight loss and general fatigue. Some lung cancers do not cause any noticeable symptoms until they are quite advanced and have spread to other parts of the body.
Q. How is lung cancer treated?
A. Cancer Healer is really effective in Lung Cancer and brings marked improvement even at the last stages. It treats lung cancer promptly without any side effects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations.
Q. How is lung cancer diagnosed?
A. Lung cancers are sometimes first detected on routine chest X-rays. However, the main method of diagnosis is bronchoscopy, in which a thin, flexible tube is inserted down the airways (under anaesthetic), allowing doctors to see the inside of the lungs and even take a biopsy (a sample small of the suspect tissue). A CT scan, liver ultrasound or bone scan may also be used to find out if the cancer has spread.
Q. Does diet affect the risk of getting lung cancer?
Q. Are there different types of lung cancer?
Q. What causes lung cancer?
Q. Does lung cancer run in families?
Q. What are the symptoms of lung cancer?
Q. How is lung cancer treated?
Q. How is lung cancer diagnosed?
Q. Does diet affect the risk of getting lung cancer?
A. This is still being investigated, but research to date has not found any link between diet and lung cancer.
Q. Are there different types of lung cancer?
A. There are four main types of lung cancer: small cell lung cancer, squamous cell carcinoma, large cell carcinoma and adenocarcinoma Tobacco smoking is strongly linked to the first three but only weakly linked to adenocarcinoma However, this type of lung cancer has been linked to the use of low-tar cigarettes.
Q. What causes lung cancer?
A. The vast majority - over 80% - of lung cancers are caused by smoking tobacco or by indirect exposure to tobacco smoke (passive smoking). The other main causes are breathing industrial chemicals such as asbestos, arsenic and polycyclic hydrocarbons or the natural radioactive gas, radon.
Q. Does lung cancer run in families?
A. There are very few, if any, inherited conditions that increase the risk of lung cancer in non-smokers. However, not all of the people who smoke get lung cancer and there may be an inherited component which influences whether or not smoking will cause lung cancer.
Q. What are the symptoms of lung cancer?
A. There are a variety of symptoms of lung cancer, including difficulty breathing, coughing up blood, chest pain, loss of appetite, weight loss and general fatigue. Some lung cancers do not cause any noticeable symptoms until they are quite advanced and have spread to other parts of the body.
Q. How is lung cancer treated?
A. Cancer Healer is really effective in Lung Cancer and brings marked improvement even at the last stages. It treats lung cancer promptly without any side effects and can also go in conjunction with chemo therapy as well as radio therapy and even after operations.
Q. How is lung cancer diagnosed?
A. Lung cancers are sometimes first detected on routine chest X-rays. However, the main method of diagnosis is bronchoscopy, in which a thin, flexible tube is inserted down the airways (under anaesthetic), allowing doctors to see the inside of the lungs and even take a biopsy (a sample small of the suspect tissue). A CT scan, liver ultrasound or bone scan may also be used to find out if the cancer has spread.
Subscribe to:
Posts (Atom)